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CagriSema leverages synergistic mechanisms: Semaglutide slows gastric emptying, suppresses glucagon, stimulates insulin, and reduces appetite, while Cagrilintide enhances satiety and further delays gastric emptying via brainstem pathways
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To improve the safety and effectiveness of UC treatment, future research needs to better understand the pathogenic mechanisms related to the inflammatory response and intestinal epithelial barrier damage in the treatment and progression of UC disease