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It provides an overview of the relevant pathways and molecular mechanisms that have been investigated in recent years, highlighting the roles of inducers and inhibitors of ferroptosis in stroke
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To test this, we over-expressed S-adenosylhomocysteine hydrolase (AHCY) in GNMT Δ8 expressing cells ( Supplementary Figure S4B ), which converts SAH to homocysteine, to lower SAH levels and determine whether this could rescue the defect
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