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78c prevents lysolecithin- and PA-mediated myelin protein and oligodendrocyte loss Since we observed protective effects of genetic CD38 inhibition on oligodendrocyte survival following HFD (Fig 2), as well as increased CD38 expression in a demyelination model (Fig 1), we next sought to explore benefits conferred by pharmacologic inhibition using 78c in a model of lysolecithin-induced ex vivo demyelination
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