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A plausible mechanism for prolonged spike protein production involves latent integration of mRNA vaccine-derived genetic material, including plasmid-sourced DNA and promoters such as SV40, into host genomes

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We reasoned that mouse models of 22q11.2DS might have metabolic disturbances similar to those reported in patients, in which case the in vivo genetic and pharmacological manipulation of the model should provide insights into the contribution of the metabolic phenotypes to the disease pathogenesis and/or disease mechanisms